Why do we age? One of the leading theories is the 'Mitochondrial Theory of Aging'. It states that the accumulation of damage to our cellular powerhouses is a major driver of the aging process. When mitochondria become inefficient, they produce more free radicals, leading to further cellular stress—a vicious cycle.
At the same time, our telomeres, the protective caps of our chromosomes, shorten, and the levels of the vital coenzyme NAD+ drop dramatically. The result: our cells 'forget' their function, and tissues begin to degenerate. But is this process set in stone?
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Scientists estimate that a 50-year-old often has only about half the mitochondrial capacity of a 20-year-old.
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Research says: No, not necessarily. We can improve the quality of our mitochondria. Caloric restriction and exercise are proven methods to activate autophagy—the cellular recycling program that breaks down old and defective cell components.
Specific precursors of NAD+ and substances like resveratrol also show promising results in studies for activating sirtuins, our 'longevity genes'. The goal is not just to live longer (Lifespan), but to stay healthy and vital for as long as possible (Healthspan). Your mitochondria play a role in this.
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